• Login
    View Item 
    •   Home
    • iBEST Institute of Biomedical and Environmental Science and Technology - to April 2016
    • Cell and Cryobiology Research Group
    • View Item
    •   Home
    • iBEST Institute of Biomedical and Environmental Science and Technology - to April 2016
    • Cell and Cryobiology Research Group
    • View Item
    JavaScript is disabled for your browser. Some features of this site may not work without it.

    Browse

    All of UOBREPCommunitiesTitleAuthorsIssue DateSubmit DateSubjectsPublisherJournalDepartmentThis CollectionTitleAuthorsIssue DateSubmit DateSubjectsPublisherJournalDepartment

    My Account

    LoginRegister

    About

    AboutLearning ResourcesResearch Graduate SchoolResearch InstitutesUniversity Website

    Statistics

    Display statistics

    TWIST1 regulates the activity of ubiquitin proteasome system via the miR-199/214 cluster in human end-stage dilated cardiomyopathy

    • CSV
    • RefMan
    • EndNote
    • BibTex
    • RefWorks
    Thumbnail
    Name:
    Publisher version
    View Source
    Access full-text PDFOpen Access
    View Source
    Check access options
    Check access options
    Authors
    Baumgarten, Anna
    Bang, Claudia
    Tschirner, Anika
    Engelmann, Anke
    Adams, Volker
    von Haehling, Stephan
    Doehner, Wolfram
    Pregla, Reinhard
    Anker, Markus S.
    Blecharz, Kinga
    Meyer, Rudolf
    Hetzer, Roland
    Anker, Stefan D.
    Thum, Thomas
    Springer, Jochen
    Show allShow less
    Affiliation
    Charité Medical School
    Medical School Hannover
    Heart Center Leipzig
    German Heart Center Berlin
    IRCCS San Raffaele
    University of East Anglia
    Issue Date
    2013-09-30
    Subjects
    TWIST1
    ubiquitin proteasome system
    dilated cardiomyopathy
    transcription
    
    Metadata
    Show full item record
    Abstract
    The transcription factor TWIST1 has been described to regulate the microRNA (miR)-199/214 cluster. Genetic disruption of TWIST1 resulted in a cachectic phenotype and early death of the knock-out mice. This might be connected to the activity of the ubiquitin-proteasome-system (UPS), as miR-199a has been suggested to regulate the ubiquitin E2 ligases Ube2i and Ube2g1.
    Citation
    Baumgarten, A. et al (2013) 'TWIST1 regulates the activity of ubiquitin proteasome system via the miR-199/214 cluster in human end-stage dilated cardiomyopathy' Int. J. Cardiol. 168 (2):1447-52
    Publisher
    Elsevier
    Journal
    International journal of cardiology
    URI
    http://hdl.handle.net/10547/593473
    DOI
    10.1016/j.ijcard.2012.12.094
    PubMed ID
    23360823
    Additional Links
    http://www.sciencedirect.com/science/article/pii/S0167527313000053
    Type
    Article
    Language
    en
    ISSN
    1874-1754
    ae974a485f413a2113503eed53cd6c53
    10.1016/j.ijcard.2012.12.094
    Scopus Count
    Collections
    Cell and Cryobiology Research Group

    entitlement

    Related articles

    • Signal transducer and activator of transcription 3-mediated regulation of miR-199a-5p links cardiomyocyte and endothelial cell function in the heart: a key role for ubiquitin-conjugating enzymes.
    • Authors: Haghikia A, Missol-Kolka E, Tsikas D, Venturini L, Brundiers S, Castoldi M, Muckenthaler MU, Eder M, Stapel B, Thum T, Haghikia A, Petrasch-Parwez E, Drexler H, Hilfiker-Kleiner D, Scherr M
    • Issue date: 2011 May
    • The Ubiquitin Proteasome System in Ischemic and Dilated Cardiomyopathy.
    • Authors: Spänig S, Kellermann K, Dieterlen MT, Noack T, Lehmann S, Borger MA, Garbade J, Barac YD, Emrich F
    • Issue date: 2019 Dec 17
    • Elevated p53 expression is associated with dysregulation of the ubiquitin-proteasome system in dilated cardiomyopathy.
    • Authors: Birks EJ, Latif N, Enesa K, Folkvang T, Luong le A, Sarathchandra P, Khan M, Ovaa H, Terracciano CM, Barton PJ, Yacoub MH, Evans PC
    • Issue date: 2008 Aug 1
    • Perturbation of cullin deneddylation via conditional Csn8 ablation impairs the ubiquitin-proteasome system and causes cardiomyocyte necrosis and dilated cardiomyopathy in mice.
    • Authors: Su H, Li J, Menon S, Liu J, Kumarapeli AR, Wei N, Wang X
    • Issue date: 2011 Jan 7
    • Heterozygous LmnadelK32 mice develop dilated cardiomyopathy through a combined pathomechanism of haploinsufficiency and peptide toxicity.
    • Authors: Cattin ME, Bertrand AT, Schlossarek S, Le Bihan MC, Skov Jensen S, Neuber C, Crocini C, Maron S, Lainé J, Mougenot N, Varnous S, Fromes Y, Hansen A, Eschenhagen T, Decostre V, Carrier L, Bonne G
    • Issue date: 2013 Aug 1
    DSpace software (copyright © 2002 - 2021)  DuraSpace
    Quick Guide | Contact Us
    Open Repository is a service operated by 
    Atmire NV
     

    Export search results

    The export option will allow you to export the current search results of the entered query to a file. Different formats are available for download. To export the items, click on the button corresponding with the preferred download format.

    By default, clicking on the export buttons will result in a download of the allowed maximum amount of items.

    To select a subset of the search results, click "Selective Export" button and make a selection of the items you want to export. The amount of items that can be exported at once is similarly restricted as the full export.

    After making a selection, click one of the export format buttons. The amount of items that will be exported is indicated in the bubble next to export format.